A second site Kras(G12D) mutation that impairs PI3K binding rescues embryonic lethality, abrogates myeloproliferative disease, and delays lung tumorigenesis.

Wong, J. C., Perez-Mancera, P. A., Kim, J., Jen, K. Y., Kogan, S. C., Firestone, A. J., Collisson, Eric A, Tuveson, D. A., Shannon, K. (May 2020) A second site Kras(G12D) mutation that impairs PI3K binding rescues embryonic lethality, abrogates myeloproliferative disease, and delays lung tumorigenesis. Molecular Cancer Research, 18 (5). p. 43. ISSN 1541-7786

Item Type:	Paper
CSHL Authors:	Tuveson, David A.
Communities:	CSHL labs > Tuveson lab
Depositing User:	Matthew Dunn
Date:	May 2020
Date Deposited:	06 Jul 2020 18:36
Last Modified:	06 Jul 2020 18:36
URI:	https://repository.cshl.edu/id/eprint/39507

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